X-linked sideroblastic anemia due to ALAS2 intron 1 enhancer element GATA-binding site mutations.

نویسندگان

  • Dean R Campagna
  • Charlotte I de Bie
  • Klaus Schmitz-Abe
  • Marion Sweeney
  • Anoop K Sendamarai
  • Paul J Schmidt
  • Matthew M Heeney
  • Helger G Yntema
  • Caroline Kannengiesser
  • Bernard Grandchamp
  • Charlotte M Niemeyer
  • Nine V A M Knoers
  • Sonia Swart
  • Gordon Marron
  • Richard van Wijk
  • Reinier A Raymakers
  • Alison May
  • Kyriacos Markianos
  • Sylvia S Bottomley
  • Dorine W Swinkels
  • Mark D Fleming
چکیده

X-linked sideroblastic anemia (XLSA) is the most common form of congenital sideroblastic anemia. In affected males, it is uniformly associated with partial loss-of-function missense mutations in the erythroid-specific heme biosynthesis protein 5-aminolevulinate synthase 2 (ALAS2). Here, we report five families with XLSA owing to mutations in a GATA transcription factor binding site located in a transcriptional enhancer element in intron 1 of the ALAS2 gene. As such, this study defines a new class of mutations that should be evaluated in patients undergoing genetic testing for a suspected diagnosis of XLSA.

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عنوان ژورنال:
  • American journal of hematology

دوره 89 3  شماره 

صفحات  -

تاریخ انتشار 2014